rabbit polyclonal antibody against insulin (Cell Signaling Technology Inc)
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Cell Signaling Technology Inc
rabbit polyclonal antibody against insulin
Rabbit Polyclonal Antibody Against Insulin, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 96/100, based on 653 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/antibodies+against+insulin/Insulin+Antibody/pm41891636-86-0-11
Average 96 stars, based on 653 article reviews
Rabbit Polyclonal Antibody Against Insulin, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 96/100, based on 653 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/antibodies+against+insulin/Insulin+Antibody/pm41891636-86-0-11
Average 96 stars, based on 653 article reviews
rabbit polyclonal antibody against insulin - by Bioz Stars,
2026-10
96/100 stars
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Immunohistochemical staining:Article Title: Effects of a Sodium-Glucose Cotransporter 2 Inhibitor Dapagliflozin on Pancreas in Obese Diabetic Mice. Article Snippet: .. Immunohistochemical examination was performed using Article Title: Effects of a Sodium-Glucose Cotransporter 2 Inhibitor Dapagliflozin on Pancreas in Obese Diabetic Mice Article Snippet: .. Immunohistochemical examination was performed using Article Title: Bone marrow mesenchymal stem cells-derived exosomes protect against β-cell destruction models and kidney injury by suppressing ferroptosis. Article Snippet: ImageJ software (NIH, USA) was used to quantify the islet-to-pancreas area (%), islet density, and average islet-positive area in the pancreas33. .. Immunohistochemical staining of pancreatic tissue was performed using Article Title: Bone marrow mesenchymal stem cells-derived exosomes protect against β-cell destruction models and kidney injury by suppressing ferroptosis Article Snippet: ImageJ software (NIH, USA) was used to quantify the islet-to-pancreas area (%), islet density, and average islet-positive area in the pancreas . .. Immunohistochemical staining of pancreatic tissue was performed using Staining:Article Title: Bone marrow mesenchymal stem cells-derived exosomes protect against β-cell destruction models and kidney injury by suppressing ferroptosis. Article Snippet: ImageJ software (NIH, USA) was used to quantify the islet-to-pancreas area (%), islet density, and average islet-positive area in the pancreas33. .. Immunohistochemical staining of pancreatic tissue was performed using Article Title: Bone marrow mesenchymal stem cells-derived exosomes protect against β-cell destruction models and kidney injury by suppressing ferroptosis Article Snippet: ImageJ software (NIH, USA) was used to quantify the islet-to-pancreas area (%), islet density, and average islet-positive area in the pancreas . .. Immunohistochemical staining of pancreatic tissue was performed using Incubation:Article Title: Vitamin D augments insulin secretion via calcium influx and upregulation of voltage calcium channels: Findings from INS-1 cells and human islets. Article Snippet: Vitamin D (VD) has been implicated in regulating insulin secretion and pancreatic β-cell function.. Yet, the underlying molecular mechanism of VD in glucose homeostasis is not fully understood.. This study investigates the effect of VD in regulating insulin secretion and pancreatic β-cell function. Article Title: Loss of Beta‐Cell Identity and Function as a Mechanism of Secondary Failure of Sulfonylurea Therapy in Diabetes Article Snippet: .. Then blocked in 3% bovine serum albumin and incubated overnight in primary Article Title: Proton activated chloride channel and its regulation of insulin secretion in β cells. Article Snippet: .. Cells were then incubated overnight at 4◦C with primary |
![Effects of Pas2r12 or Pas2r12–cargo protein complex-mediated stimulation on <t>INSR/IGF1R</t> and ERK1/2. HEK293 cells were pretreated with dimethyl sulfoxide (linsitinib [−]) or with linsitinib (linsitinib [+]) and subsequently stimulated with Pas2r12, Pas2r12–EGFP, Pas2r12–IgG, or insulin for 2 min ( A – C ) or 10 min ( D – F ). Panels A and D show representative Western blot images. Panels B and E display the phosphorylation levels of INSR/IGF1R (pINSR/pIGF1R); panels C and F show the phosphorylation levels of ERK1/2 (pERK1/2); all values were normalized to GAPDH. Phosphorylation levels were analyzed and compared with the solvent control (DMEM only, linsitinib [−]) using Student’s t -test. For each treatment condition, linsitinib (+) was compared with the corresponding linsitinib (−) condition. Statistically significant differences compared with the control are indicated on the bar graphs; significant differences between linsitinib (+) and linsitinib (−) treatments are marked by horizontal lines. Statistical comparisons were performed against control cells using Student’s t -test. Error bars indicate the standard error of the mean (SEM). * p < 0.05 and ** p < 0.01. N = 4.](https://pub-med-central-images-cdn.bioz.com/pub_med_central_ids_ending_with_6253/pmc12736253/pmc12736253__pharmaceuticals-18-01885-g001.jpg)
![Effects of Pas2r12 or Pas2r12–cargo protein complex stimulation on INSR/IGF1R in IGF1R-overexpressing cells. ( A ) HEKI#66 cells pretreated with DMSO (linsitinib [−]) or linsitinib (linsitinib [+]) were stimulated with Pas2r12, Pas2r12–EGFP, or insulin for 2 min. Level of phosphorylated INSR/IGF1R (pINSR/pIGF1R) was assessed by Western blot. ( B ) Relative levels of pINSR/pIGF1R, normalized to GAPDH, corresponding to the data in panel A. Phosphorylation levels were analyzed using Student’s t -test. Each treatment condition was compared with the solvent-treated HEKI#66 control (linsitinib [−]), and, for each treatment, phosphorylation levels in the linsitinib (−) and linsitinib (+) conditions were also compared. Statistically significant differences compared with the solvent-treated control are indicated on the bar graphs, and significant differences between linsitinib (+) and linsitinib (−) treatments are marked by horizontal lines. Error bars indicate the standard error of the mean (SEM). * p < 0.05, *** p < 0.001. N = 3.](https://pub-med-central-images-cdn.bioz.com/pub_med_central_ids_ending_with_6253/pmc12736253/pmc12736253__pharmaceuticals-18-01885-g005.jpg)
![Effects of Pas2r12 or Pas2r12–cargo protein complex stimulation on INSR/IGF1R in INSR-overexpressing cells. ( A ) IN#1 cells pretreated with DMSO (linsitinib [−]) or linsitinib (linsitinib [+]) were stimulated with Pas2r12, Pas2r12–EGFP, or insulin for 2 min. Phosphorylation of INSR/IGF1R (pINSR/pIGF1R) was assessed by Western blot. ( B ) Relative levels of pINSR/pIGF1R were normalized to GAPDH, corresponding to the data in panel A. Phosphorylation levels were analyzed using Student’s t -test. Each treatment condition was compared with the solvent-treated IN#1 control (linsitinib [−]), and for each treatment, phosphorylation levels in the linsitinib (−) and linsitinib (+) conditions were also compared. Statistically significant differences compared with the solvent-treated control are indicated on the bar graphs, and significant differences between linsitinib (+) and linsitinib (−) treatments are marked by horizontal lines. Error bars indicate the standard error of the mean (SEM). * p < 0.05, ** p < 0.01, and *** p < 0.001. N = 3.](https://pub-med-central-images-cdn.bioz.com/pub_med_central_ids_ending_with_6253/pmc12736253/pmc12736253__pharmaceuticals-18-01885-g008.jpg)
